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Research at the OSU College of Veterinary Medicine is dedicated to solving some of the world's most challenging animal and human health issues.
CVM research focuses on the development of animal models to study human diseases like diabetes, HIV, and tuberculosis. Our approach is interdisciplinary and often involves collaboration with other colleges on campus. Trained in comparative biology, our veterinary scientists are able to work with colleagues in a wide arrary of disciplines to explore solutions to complex problems involving the interfaces between biosciences, physical sciences, social sciences, and engineering.
This collaborative spirit is essential for the education of our doctoral students, many of whom are enrolled in the interdepartmental Molecular and Cell Biology program. In addition to doctoral programs, the college provides research laboratory experiences for professional veterinary students and undergraduate students from other colleges on campus.
The OSU College of Veterinary Medicine has leveraged this interdisciplinary approach to create a critical mass of expertise and resources that allow us to compete successfully for funding, support high-quality graduate education, build state-of-the-art facilities, and establish a world-class reputation for research excellence.
Find out more about our signature areas of research.
- Histopathological features of canine spontaneous non-neoplastic gastric polyps - a retrospective study of 15 cases.
- Placement of a caudal vena cava stent for treatment of Budd-Chiari-like syndrome in a 4-month-old Ragdoll cat.
- Neurotropic T-cell-rich B-cell lymphoma in a 14-year-old Morgan gelding.
- Culture of equine fibroblast-like synoviocytes on synthetic tissue scaffolds towards meniscal tissue engineering: a preliminary cell-seeding study.
- Identification of Mycobacterium avium genes associated with resistance to host antimicrobial peptides.
- Intercostal approach for right adrenalectomy in dogs.
- Mycobacterium avium biofilm attenuates mononuclear phagocyte function by triggering hyperstimulation and apoptosis during early infection.