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Research at the OSU College of Veterinary Medicine is dedicated to solving some of the world's most challenging animal and human health issues.
CVM research focuses on the development of animal models to study human diseases like diabetes, HIV, and tuberculosis. Our approach is interdisciplinary and often involves collaboration with other colleges on campus. Trained in comparative biology, our veterinary scientists are able to work with colleagues in a wide arrary of disciplines to explore solutions to complex problems involving the interfaces between biosciences, physical sciences, social sciences, and engineering.
This collaborative spirit is essential for the education of our doctoral students, many of whom are enrolled in the interdepartmental Molecular and Cell Biology program. In addition to doctoral programs, the college provides research laboratory experiences for professional veterinary students and undergraduate students from other colleges on campus.
The OSU College of Veterinary Medicine has leveraged this interdisciplinary approach to create a critical mass of expertise and resources that allow us to compete successfully for funding, support high-quality graduate education, build state-of-the-art facilities, and establish a world-class reputation for research excellence.
Find out more about our signature areas of research.
- Country- and age-specific optimal allocation of dengue vaccines.
- Autolysis in Vibrio tubiashii and Vibrio coralliilyticus.
- Pathology in practice. Primitive neuroectodermal tumor (PNET) with ependymal differentiation.
- Seasonal effects of heat shock on bacterial populations, including artificial Vibrio parahaemolyticus exposure, in the Pacific oyster, Crassostrea gigas.
- A multiplex biomarker approach for the diagnosis of transitional cell carcinoma from canine urine.
- Roles of the sodium-translocating NADH:quinone oxidoreductase (Na+-NQR) on vibrio cholerae metabolism, motility and osmotic stress resistance.
- Effect of combined systemic and local morpholino treatment on the spinal muscular atrophy Δ7 mouse model phenotype.