Found 9 results
Author [ Title(Desc)] Type Year
Filters: Author is Osorio, Nelson  [Clear All Filters]
A B C D E F G H I J K L M N O P Q R S T U V W X Y Z 
C
L. Jin, Carpenter, D., Moerdyk-Schauwecker, M., Vanarsdall, A. L., Osorio, N., Hsiang, C., Jones, C., and Wechsler, S. L., “Cellular FLIP can substitute for the herpes simplex virus type 1 latency-associated transcript gene to support a wild-type virus reactivation phenotype in mice.”, Journal of neurovirology, vol. 14, no. 5, pp. 389-400, 2008.
G
G. - C. Perng, Maguen, B., Jin, L., Mott, K. R., Osorio, N., Slanina, S. M., Yukht, A., Ghiasi, H., Nesburn, A. B., Inman, M., Henderson, G., Jones, C., and Wechsler, S. L., “A gene capable of blocking apoptosis can substitute for the herpes simplex virus type 1 latency-associated transcript gene and restore wild-type reactivation levels.”, Journal of virology, vol. 76, no. 3, pp. 1224-35, 2002.
H
J. Naito, Mott, K. R., Osorio, N., Jin, L., and Perng, G. - C., “Herpes simplex virus type 1 immediate-early protein ICP0 diffuses out of infected rabbit corneas.”, The Journal of general virology, vol. 86, no. Pt 11, pp. 2979-88, 2005.
L. Jin, Perng, G. - C., Mott, K. R., Osorio, N., Naito, J., Brick, D. J., Carpenter, D., Jones, C., and Wechsler, S. L., “A herpes simplex virus type 1 mutant expressing a baculovirus inhibitor of apoptosis gene in place of latency-associated transcript has a wild-type reactivation phenotype in the mouse.”, Journal of virology, vol. 79, no. 19, pp. 12286-95, 2005.
M
D. Chan, Cohen, J., Naito, J., Mott, K. R., Osorio, N., Jin, L., Fraser, N. W., Jones, C., Wechsler, S. L., and Perng, G. Chuen, “A mutant deleted for most of the herpes simplex virus type 1 (HSV-1) UOL gene does not affect the spontaneous reactivation phenotype in rabbits.”, Journal of neurovirology, vol. 12, no. 1, pp. 5-16, 2006.
N
G. - C. Perng, Maguen, B., Jin, L., Mott, K. R., Kurylo, J., BenMohamed, L., Yukht, A., Osorio, N., Nesburn, A. B., Henderson, G., Inman, M., Jones, C., and Wechsler, S. L., “A novel herpes simplex virus type 1 transcript (AL-RNA) antisense to the 5' end of the latency-associated transcript produces a protein in infected rabbits.”, Journal of virology, vol. 76, no. 16, pp. 8003-10, 2002.
R
L. Jin, Perng, G. - C., Carpenter, D., Mott, K. R., Osorio, N., Naito, J., Brick, D. J., Jones, C., and Wechsler, S. L., “Reactivation phenotype in rabbits of a herpes simplex virus type 1 mutant containing an unrelated antiapoptosis gene in place of latency-associated transcript.”, Journal of neurovirology, vol. 13, no. 1, pp. 78-84, 2007.
S
D. Carpenter, Singh, S., Osorio, N., Hsiang, C., Jiang, X., Jin, L., Jones, C., and Wechsler, S. L., “A speculated ribozyme site in the herpes simplex virus type 1 latency-associated transcript gene is not essential for a wild-type reactivation phenotype.”, Journal of neurovirology, vol. 14, no. 6, pp. 558-62, 2008.
D. Carpenter, Hsiang, C., Brown, D. J., Jin, L., Osorio, N., BenMohamed, L., Jones, C., and Wechsler, S. L., “Stable cell lines expressing high levels of the herpes simplex virus type 1 LAT are refractory to caspase 3 activation and DNA laddering following cold shock induced apoptosis.”, Virology, vol. 369, no. 1, pp. 12-8, 2007.