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R. G. Bastos, Sears, K. P., Dinkel, K. D., Knowles, D. P., and Fry, L. M., Changes in the Molecular and Functional Phenotype of Bovine Monocytes during Infection., Infect Immun, vol. 87, no. 12, 2019.
L. E. Bermudez, Petrofsky, M., Wu, M., and Young, L. S., Clarithromycin significantly improves interleukin-12-mediated anti-Mycobacterium avium activity and abolishes toxicity in mice., The Journal of infectious diseases, vol. 178, no. 3, pp. 896-9, 1998.
L. E. Bermudez and Young, L. S., Tumor necrosis factor alpha stimulates mycobactericidal/mycobacteriostatic activity in human macrophages by a protein kinase C-independent pathway., Cellular immunology, vol. 144, no. 2, pp. 258-68, 1992.
L. E. Bermudez and Young, L. S., Killing of Mycobacterium avium: insights provided by the use of recombinant cytokines., Research in microbiology, vol. 141, no. 2, pp. 241-3, 1990.
L. E. Bermudez, Inderlied, C., and Young, L. S., Stimulation with cytokines enhances penetration of azithromycin into human macrophages., Antimicrobial agents and chemotherapy, vol. 35, no. 12, pp. 2625-9, 1991.
L. E. Bermudez and Young, L. S., Activities of amikacin, roxithromycin, and azithromycin alone or in combination with tumor necrosis factor against Mycobacterium avium complex., Antimicrobial agents and chemotherapy, vol. 32, no. 8, pp. 1149-53, 1988.
L. E. Bermudez, Parker, A., and Petrofsky, M., Apoptosis of Mycobacterium avium-infected macrophages is mediated by both tumour necrosis factor (TNF) and Fas, and involves the activation of caspases., Clinical and experimental immunology, vol. 116, no. 1, pp. 94-9, 1999.
L. E. Bermudez, Differential mechanisms of intracellular killing of Mycobacterium avium and Listeria monocytogenes by activated human and murine macrophages. The role of nitric oxide., Clinical and experimental immunology, vol. 91, no. 2, pp. 277-81, 1993.
L. E. Bermudez and Young, L. S., Recombinant granulocyte-macrophage colony-stimulating factor activates human macrophages to inhibit growth or kill Mycobacterium avium complex., Journal of leukocyte biology, vol. 48, no. 1, pp. 67-73, 1990.
L. E. Bermudez, Wu, M., and Young, L. S., Interleukin-12-stimulated natural killer cells can activate human macrophages to inhibit growth of Mycobacterium avium., Infection and immunity, vol. 63, no. 10, pp. 4099-104, 1995.
L. E. Bermudez and Young, L. S., Ethanol augments intracellular survival of Mycobacterium avium complex and impairs macrophage responses to cytokines., The Journal of infectious diseases, vol. 163, no. 6, pp. 1286-92, 1991.
L. E. Bermudez and Young, L. S., Tumor necrosis factor, alone or in combination with IL-2, but not IFN-gamma, is associated with macrophage killing of Mycobacterium avium complex., Journal of immunology (Baltimore, Md. : 1950), vol. 140, no. 9, pp. 3006-13, 1988.
L. E. Bermudez, Production of transforming growth factor-beta by Mycobacterium avium-infected human macrophages is associated with unresponsiveness to IFN-gamma., Journal of immunology (Baltimore, Md. : 1950), vol. 150, no. 5, pp. 1838-45, 1993.
L. E. Bermudez, Young, L. S., and Gupta, S., 1,25 Dihydroxyvitamin D3-dependent inhibition of growth or killing of Mycobacterium avium complex in human macrophages is mediated by TNF and GM-CSF., Cellular immunology, vol. 127, no. 2, pp. 432-41, 1990.
L. E. Bermudez, Wu, M., Martinelli, J., and Young, L. S., Ethanol affects release of TNF and GM-CSF and membrane expression of TNF receptors by human macrophages., Lymphokine and cytokine research, vol. 10, no. 5, pp. 413-9, 1991.
L. E. Bermudez, Stevens, P., Kolonoski, P., Wu, M., and Young, L. S., Treatment of experimental disseminated Mycobacterium avium complex infection in mice with recombinant IL-2 and tumor necrosis factor., Journal of immunology (Baltimore, Md. : 1950), vol. 143, no. 9, pp. 2996-3000, 1989.
L. E. Bermudez, Wu, M., Miltner, E., and Inderlied, C. B., Isolation of two subpopulations of Mycobacterium avium within human macrophages., FEMS microbiology letters, vol. 178, no. 1, pp. 19-26, 1999.
L. E. Bermudez, Covaro, G., and Remington, J., Infection of murine macrophages with Toxoplasma gondii is associated with release of transforming growth factor beta and downregulation of expression of tumor necrosis factor receptors., Infection and immunity, vol. 61, no. 10, pp. 4126-30, 1993.
L. E. Bermudez, Parker, A., and Goodman, J. R., Growth within macrophages increases the efficiency of Mycobacterium avium in invading other macrophages by a complement receptor-independent pathway., Infection and immunity, vol. 65, no. 5, pp. 1916-25, 1997.
L. E. Bermudez, Wu, M., Petrofsky, M., and Young, L. S., Interleukin-6 antagonizes tumor necrosis factor-mediated mycobacteriostatic and mycobactericidal activities in macrophages., Infection and immunity, vol. 60, no. 10, pp. 4245-52, 1992.
L. E. Bermudez and Young, L. S., Ethanol and survival of Mycobacterium avium complex within macrophages., Progress in clinical and biological research, vol. 325, pp. 383-91, 1990.
L. E. Bermudez, Wu, M., and Young, L. S., Effect of stress-related hormones on macrophage receptors and response to tumor necrosis factor., Lymphokine research, vol. 9, no. 2, pp. 137-45, 1990.
C. M. Black, Bermudez, L. E., Young, L. S., and Remington, J. S., Co-infection of macrophages modulates interferon gamma and tumor necrosis factor-induced activation against intracellular pathogens., The Journal of experimental medicine, vol. 172, no. 3, pp. 977-80, 1990.

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