Journal Article
Y. Yamazaki, Danelishvili, L., Wu, M., Hidaka, E., Katsuyama, T., Stang, B., Petrofsky, M., Bildfell, R., and Bermudez, L. E.,
“The ability to form biofilm influences Mycobacterium avium invasion and translocation of bronchial epithelial cells.”,
Cell Microbiol, vol. 8, no. 5, pp. 806-14, 2006.
Y. Yamazaki, Danelishvili, L., Wu, M., Hidaka, E., Katsuyama, T., Stang, B. V., Petrofsky, M., Bildfell, R. J., and Bermudez, L. E.,
“The ability to form biofilm influences Mycobacterium avium invasion and translocation of bronchial epithelial cells.”,
Cellular microbiology, vol. 8, no. 5, pp. 806-14, 2006.
L. E. Bermudez, Sangari, F. J., Kolonoski, P., Petrofsky, M., and Goodman, J.,
“The efficiency of the translocation of Mycobacterium tuberculosis across a bilayer of epithelial and endothelial cells as a model of the alveolar wall is a consequence of transport within mononuclear phagocytes and invasion of alveolar epithelial cells.”,
Infection and immunity, vol. 70, no. 1, pp. 140-6, 2002.
L. E. Bermudez, Inderlied, C. B., Kolonoski, P., Chee, C. B., Aralar, P., Petrofsky, M., Parman, T., Green, C. E., Lewin, A. H., Ellis, W. Y., and Young, L. S.,
“Identification of (+)-Erythro-Mefloquine as an Active Enantiomer with Greater Efficacy than Mefloquine against Mycobacterium avium Infection in Mice.”,
Antimicrobial agents and chemotherapy, vol. 56, no. 8, pp. 4202-6, 2012.
Y. -jun Li, Danelishvili, L., Wagner, D., Petrofsky, M., and Bermudez, L. E.,
“Identification of virulence determinants of Mycobacterium avium that impact on the ability to resist host killing mechanisms.”,
J Med Microbiol, vol. 59, no. Pt 1, pp. 8-16, 2010.
Y. -jun Li, Danelishvili, L., Wagner, D., Petrofsky, M., and Bermudez, L. E.,
“Identification of virulence determinants of Mycobacterium avium that impact on the ability to resist host killing mechanisms.”,
Journal of medical microbiology, vol. 59, no. Pt 1, pp. 8-16, 2010.
L. E. Bermudez, Petrofsky, M., and Sangari, F.,
“Intracellular phenotype of Mycobacterium avium enters macrophages primarily by a macropinocytosis-like mechanism and survives in a compartment that differs from that with extracellular phenotype.”,
Cell biology international, vol. 28, no. 5, pp. 411-9, 2004.
L. E. Bermudez, Kolonoski, P., Petrofsky, M., Wu, M., Inderlied, C. B., and Young, L. S.,
“Mefloquine, moxifloxacin, and ethambutol are a triple-drug alternative to macrolide-containing regimens for treatment of Mycobacterium avium disease.”,
The Journal of infectious diseases, vol. 187, no. 12, pp. 1977-80, 2003.
D. Wagner, Sangari, F. J., Kim, S., Petrofsky, M., and Bermudez, L. E.,
“Mycobacterium avium infection of macrophages results in progressive suppression of interleukin-12 production in vitro and in vivo.”,
Journal of leukocyte biology, vol. 71, no. 1, pp. 80-8, 2002.
Y. Li, Miltner, E., Wu, M., Petrofsky, M., and Bermudez, L. E.,
“A Mycobacterium avium PPE gene is associated with the ability of the bacterium to grow in macrophages and virulence in mice.”,
Cellular microbiology, vol. 7, no. 4, pp. 539-48, 2005.
Y. -jun Li, Petrofsky, M., and Bermudez, L. E.,
“Mycobacterium tuberculosis uptake by recipient host macrophages is influenced by environmental conditions in the granuloma of the infectious individual and is associated with impaired production of interleukin-12 and tumor necrosis factor alpha.”,
Infection and immunity, vol. 70, no. 11, pp. 6223-30, 2002.
L. E. Bermudez, Petrofsky, M., Sommer, S., and Barletta, R. G.,
“Peyer's patch-deficient mice demonstrate that Mycobacterium avium subsp. paratuberculosis translocates across the mucosal barrier via both M cells and enterocytes but has inefficient dissemination.”,
Infection and immunity, vol. 78, no. 8, pp. 3570-7, 2010.
L. E. Bermudez, Kolonoski, P., Seitz, L. E., Petrofsky, M., Reynolds, R., Wu, M., and Young, L. S.,
“SRI-286, a thiosemicarbazole, in combination with mefloquine and moxifloxacin for treatment of murine Mycobacterium avium complex disease.”,
Antimicrobial agents and chemotherapy, vol. 48, no. 9, pp. 3556-8, 2004.