Found 1433 results
[ Author(Asc)] Title Type Year
A B C D E F G H I J K L M N O P Q R S T U V W X Y Z 
B
L. E. Bermudez, [Bacteremias in immunosuppressed patients]., Revista paulista de medicina, vol. 104, no. 2, pp. 87-92, 1986.
L. E. Bermudez and Young, L. S., Tumor necrosis factor alpha stimulates mycobactericidal/mycobacteriostatic activity in human macrophages by a protein kinase C-independent pathway., Cellular immunology, vol. 144, no. 2, pp. 258-68, 1992.
L. E. Bermudez, Petrofsky, M., Sommer, S., and Barletta, R. G., Peyer's patch-deficient mice demonstrate that Mycobacterium avium subsp. paratuberculosis translocates across the mucosal barrier via both M cells and enterocytes but has inefficient dissemination., Infection and immunity, vol. 78, no. 8, pp. 3570-7, 2010.
L. E. Bermudez and Kaplan, G., Recombinant cytokines for controlling mycobacterial infections., Trends in microbiology, vol. 3, no. 1, pp. 22-7, 1995.
L. E. Bermudez, Young, L. S., and Enkel, H., Interaction of Mycobacterium avium complex with human macrophages: roles of membrane receptors and serum proteins., Infection and immunity, vol. 59, no. 5, pp. 1697-702, 1991.
L. E. Bermudez, Martinelli, J., Petrofsky, M., Kolonoski, P., and Young, L. S., Recombinant granulocyte-macrophage colony-stimulating factor enhances the effects of antibiotics against Mycobacterium avium complex infection in the beige mouse model., The Journal of infectious diseases, vol. 169, no. 3, pp. 575-80, 1994.
L. E. Bermudez and Young, L. S., Recombinant granulocyte-macrophage colony-stimulating factor activates human macrophages to inhibit growth or kill Mycobacterium avium complex., Journal of leukocyte biology, vol. 48, no. 1, pp. 67-73, 1990.
L. E. Bermudez, Petrofsky, M., Kolonoski, P., and Young, L. S., Emergence of Mycobacterium avium populations resistant to macrolides during experimental chemotherapy., Antimicrobial agents and chemotherapy, vol. 42, no. 1, pp. 180-3, 1998.
L. E. Bermudez and Young, L. S., Activities of amikacin, roxithromycin, and azithromycin alone or in combination with tumor necrosis factor against Mycobacterium avium complex., Antimicrobial agents and chemotherapy, vol. 32, no. 8, pp. 1149-53, 1988.
L. E. Bermudez and Petrofsky, M., Regulation of the expression of Mycobacterium avium complex proteins differs according to the environment within host cells., Immunology and cell biology, vol. 75, no. 1, pp. 35-40, 1997.
L. E. Bermudez, Production of transforming growth factor-beta by Mycobacterium avium-infected human macrophages is associated with unresponsiveness to IFN-gamma., Journal of immunology (Baltimore, Md. : 1950), vol. 150, no. 5, pp. 1838-45, 1993.
L. E. Bermudez, Nash, K., Petrofsky, M., Young, L. S., and Inderlied, C. B., Clarithromycin-resistant mycobacterium avium is still susceptible to treatment with clarithromycin and is virulent in mice., Antimicrobial agents and chemotherapy, vol. 44, no. 10, pp. 2619-22, 2000.
L. E. Bermudez, Kolonoski, P., and Young, L. S., Roxithromycin alone and in combination with either ethambutol or levofloxacin for disseminated Mycobacterium avium infections in beige mice., Antimicrobial agents and chemotherapy, vol. 40, no. 4, pp. 1033-5, 1996.
L. E. Bermudez, Petrofsky, M., Kolonoski, P., and Young, L. S., An animal model of Mycobacterium avium complex disseminated infection after colonization of the intestinal tract., The Journal of infectious diseases, vol. 165, no. 1, pp. 75-9, 1992.
L. E. Bermudez, Wagner, D., and Sosnowska, D., Mechanisms of Mycobacterium avium pathogenesis., Archivum immunologiae et therapiae experimentalis, vol. 48, no. 6, pp. 521-7, 2000.
L. E. Bermudez, Immunobiology of Mycobacterium avium infection., European journal of clinical microbiology & infectious diseases : official publication of the European Society of Clinical Microbiology, vol. 13, no. 11, pp. 1000-6, 1994.
L. E. Bermudez, Sangari, F. J., Kolonoski, P., Petrofsky, M., and Goodman, J., The efficiency of the translocation of Mycobacterium tuberculosis across a bilayer of epithelial and endothelial cells as a model of the alveolar wall is a consequence of transport within mononuclear phagocytes and invasion of alveolar epithelial cells., Infection and immunity, vol. 70, no. 1, pp. 140-6, 2002.
L. E. Bermudez, Wu, M., and Young, L. S., Effect of stress-related hormones on macrophage receptors and response to tumor necrosis factor., Lymphokine research, vol. 9, no. 2, pp. 137-45, 1990.
L. E. Bermudez, Kolonoski, P., Wu, M., Aralar, P. A., Inderlied, C. B., and Young, L. S., Mefloquine is active in vitro and in vivo against Mycobacterium avium complex., Antimicrobial agents and chemotherapy, vol. 43, no. 8, pp. 1870-4, 1999.
L. E. Bermudez, Kemper, C. A., and Deresinski, S. C., Dysfunctional monocytes from a patient with disseminated Mycobacterium kansasii infection are activated in vitro and in vivo by GM-CSF., Biotherapy (Dordrecht, Netherlands), vol. 8, no. 2, pp. 135-42, 1994.
L. E. Bermudez and Young, L. S., Oxidative and non-oxidative intracellular killing of Mycobacterium avium complex., Microbial pathogenesis, vol. 7, no. 4, pp. 289-98, 1989.
L. E. Bermudez, Petrofsky, M., and Goodman, J., Exposure to low oxygen tension and increased osmolarity enhance the ability of Mycobacterium avium to enter intestinal epithelial (HT-29) cells., Infection and immunity, vol. 65, no. 9, pp. 3768-73, 1997.
L. E. Bermudez and Young, L. S., Phagocytosis and intracellular killing of Mycobacterium avium complex by human and murine macrophages., Brazilian journal of medical and biological research = Revista brasileira de pesquisas médicas e biológicas / Sociedade Brasileira de Biofísica .. [et al.], vol. 20, no. 2, pp. 191-201, 1987.
L. E. Bermudez, Inderlied, C. B., Kolonoski, P., Wu, M., Barbara-Burnham, L., and Young, L. S., Activities of bay Y 3118, levofloxacin, and ofloxacin alone or in combination with ethambutol against Mycobacterium avium complex in vitro, in human macrophages, and in beige mice., Antimicrobial agents and chemotherapy, vol. 40, no. 3, pp. 546-51, 1996.
L. E. Bermudez, Shelton, K., and Young, L. S., Comparison of the ability of Mycobacterium avium, M. smegmatis and M. tuberculosis to invade and replicate within HEp-2 epithelial cells., Tubercle and lung disease : the official journal of the International Union against Tuberculosis and Lung Disease, vol. 76, no. 3, pp. 240-7, 1995.

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