Found 36 results
Author Title [ Type(Desc)] Year
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Journal Article
W. Peng, Jin, L., Henderson, G., Perng, G. C., Brick, D. J., Nesburn, A. B., Wechsler, S. L., and Jones, C., “Mapping herpes simplex virus type 1 latency-associated transcript sequences that protect from apoptosis mediated by a plasmid expressing caspase-8.”, Journal of neurovirology, vol. 10, no. 4, pp. 260-5, 2004.
B. L. Brim, Haskell, R., Awedikian, R., Ellinwood, N. M., Jin, L., Kumar, A., Foster, T. C., and Magnusson, K. R., “Memory in aged mice is rescued by enhanced expression of the GluN2B subunit of the NMDA receptor.”, Behavioural brain research, vol. 238, pp. 211-26, 2013.
B. L. Brim, Haskell, R., Awedikian, R., Ellinwood, N. M., Jin, L., Kumar, A., Foster, T. C., and Magnusson, K. R., “Memory in aged mice is rescued by enhanced expression of the GluN2B subunit of the NMDA receptor.”, Behavioural brain research, vol. 238, pp. 211-26, 2013.
L. Jin, Perng, G. - C., Brick, D. J., Naito, J., Nesburn, A. B., Jones, C., and Wechsler, S. L., “Methods for detecting the HSV-1 LAT anti-apoptosis activity in virus infected tissue culture cells.”, Journal of virological methods, vol. 118, no. 1, pp. 9-13, 2004.
D. Chan, Cohen, J., Naito, J., Mott, K. R., Osorio, N., Jin, L., Fraser, N. W., Jones, C., Wechsler, S. L., and Perng, G. Chuen, “A mutant deleted for most of the herpes simplex virus type 1 (HSV-1) UOL gene does not affect the spontaneous reactivation phenotype in rabbits.”, Journal of neurovirology, vol. 12, no. 1, pp. 5-16, 2006.
G. - C. Perng, Maguen, B., Jin, L., Mott, K. R., Kurylo, J., BenMohamed, L., Yukht, A., Osorio, N., Nesburn, A. B., Henderson, G., Inman, M., Jones, C., and Wechsler, S. L., “A novel herpes simplex virus type 1 transcript (AL-RNA) antisense to the 5' end of the latency-associated transcript produces a protein in infected rabbits.”, Journal of virology, vol. 76, no. 16, pp. 8003-10, 2002.
L. Jin, Perng, G. - C., Carpenter, D., Mott, K. R., Osorio, N., Naito, J., Brick, D. J., Jones, C., and Wechsler, S. L., “Reactivation phenotype in rabbits of a herpes simplex virus type 1 mutant containing an unrelated antiapoptosis gene in place of latency-associated transcript.”, Journal of neurovirology, vol. 13, no. 1, pp. 78-84, 2007.
G. Watson, Xu, W., Reed, A., Babra, B., Putman, T., Wick, E., Wechsler, S. L., Rohrmann, G. F., and Jin, L., “Sequence and comparative analysis of the genome of HSV-1 strain McKrae.”, Virology, vol. 433, no. 2, pp. 528-37, 2012.
D. Carpenter, Singh, S., Osorio, N., Hsiang, C., Jiang, X., Jin, L., Jones, C., and Wechsler, S. L., “A speculated ribozyme site in the herpes simplex virus type 1 latency-associated transcript gene is not essential for a wild-type reactivation phenotype.”, Journal of neurovirology, vol. 14, no. 6, pp. 558-62, 2008.
D. Carpenter, Hsiang, C., Brown, D. J., Jin, L., Osorio, N., BenMohamed, L., Jones, C., and Wechsler, S. L., “Stable cell lines expressing high levels of the herpes simplex virus type 1 LAT are refractory to caspase 3 activation and DNA laddering following cold shock induced apoptosis.”, Virology, vol. 369, no. 1, pp. 12-8, 2007.
G. C. Perng, Esmaili, D., Slanina, S. M., Yukht, A., Ghiasi, H., Osorio, N., Mott, K. R., Maguen, B., Jin, L., Nesburn, A. B., and Wechsler, S. L., “Three herpes simplex virus type 1 latency-associated transcript mutants with distinct and asymmetric effects on virulence in mice compared with rabbits.”, Journal of virology, vol. 75, no. 19, pp. 9018-28, 2001.

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