B
L. E. Bermudez, Wu, M., Martinelli, J., and Young, L. S.,
“Ethanol affects release of TNF and GM-CSF and membrane expression of TNF receptors by human macrophages.”,
Lymphokine and cytokine research, vol. 10, no. 5, pp. 413-9, 1991.
L. E. Bermudez, Shelton, K., and Young, L. S.,
“Comparison of the ability of Mycobacterium avium, M. smegmatis and M. tuberculosis to invade and replicate within HEp-2 epithelial cells.”,
Tubercle and lung disease : the official journal of the International Union against Tuberculosis and Lung Disease, vol. 76, no. 3, pp. 240-7, 1995.
L. E. Bermudez, Yau-Young, A. O., Lin, J. P., Cogger, J., and Young, L. S.,
“Treatment of disseminated Mycobacterium avium complex infection of beige mice with liposome-encapsulated aminoglycosides.”,
The Journal of infectious diseases, vol. 161, no. 6, pp. 1262-8, 1990.
L. E. Bermudez, Kolonoski, P., Petrofsky, M., Wu, M., Inderlied, C. B., and Young, L. S.,
“Mefloquine, moxifloxacin, and ethambutol are a triple-drug alternative to macrolide-containing regimens for treatment of Mycobacterium avium disease.”,
The Journal of infectious diseases, vol. 187, no. 12, pp. 1977-80, 2003.
L. E. Bermudez, Kolonoski, P., Young, L. S., and Inderlied, C. B.,
“Activity of KRM 1648 alone or in combination with ethambutol or clarithromycin against Mycobacterium avium in beige mouse model of disseminated infection.”,
Antimicrobial agents and chemotherapy, vol. 38, no. 8, pp. 1844-8, 1994.
L. E. Bermudez and Young, L. S.,
“Tumor necrosis factor, alone or in combination with IL-2, but not IFN-gamma, is associated with macrophage killing of Mycobacterium avium complex.”,
Journal of immunology (Baltimore, Md. : 1950), vol. 140, no. 9, pp. 3006-13, 1988.
L. E. Bermudez, Panza, M., Velasco, E., and Vidal, E.,
“[Effect of systemic antibiotic therapy on microbial flora of gastrointestinal tract in the hematological patients].”,
Revista paulista de medicina, vol. 102, no. 6, pp. 256-9, 1984.
L. E. Bermudez, Petrofsky, M., Wu, M., and Young, L. S.,
“Clarithromycin significantly improves interleukin-12-mediated anti-Mycobacterium avium activity and abolishes toxicity in mice.”,
The Journal of infectious diseases, vol. 178, no. 3, pp. 896-9, 1998.
L. E. Bermudez, Petrofsky, M., and Shelton, K.,
“Epidermal growth factor-binding protein in Mycobacterium avium and Mycobacterium tuberculosis: a possible role in the mechanism of infection.”,
Infection and immunity, vol. 64, no. 8, pp. 2917-22, 1996.
L. E. Bermudez, Motamedi, N., Kolonoski, P., Chee, C., Baimukanova, G., Bildfell, R. J., Wang, G., Phan, L. Tam, and S Lowell, Y.,
“The efficacy of clarithromycin and the bicyclolide EDP-420 against Mycobacterium avium in a mouse model of pulmonary infection.”,
The Journal of infectious diseases, vol. 197, no. 11, pp. 1506-10, 2008.
L. E. Bermudez, Danelishvili, L., Babrack, L., and Pham, T.,
“Evidence for genes associated with the ability of Mycobacterium avium subsp. hominissuis to escape apoptotic macrophages.”,
Front Cell Infect Microbiol, vol. 5, p. 63, 2015.
L. E. Bermudez, Kolonoski, P., and Young, L. S.,
“Natural killer cell activity and macrophage-dependent inhibition of growth or killing of Mycobacterium avium complex in a mouse model.”,
Journal of leukocyte biology, vol. 47, no. 2, pp. 135-41, 1990.
L. E. Bermudez, Sangari, F. J., Kolonoski, P., Petrofsky, M., and Goodman, J.,
“The efficiency of the translocation of Mycobacterium tuberculosis across a bilayer of epithelial and endothelial cells as a model of the alveolar wall is a consequence of transport within mononuclear phagocytes and invasion of alveolar epithelial cells.”,
Infection and immunity, vol. 70, no. 1, pp. 140-6, 2002.
L. E. Bermudez, Covaro, G., and Remington, J.,
“Infection of murine macrophages with Toxoplasma gondii is associated with release of transforming growth factor beta and downregulation of expression of tumor necrosis factor receptors.”,
Infection and immunity, vol. 61, no. 10, pp. 4126-30, 1993.
L. E. Bermudez and Martins, C. A.,
“[Non-traumatic Clostridium infections].”,
AMB : revista da Associação Médica Brasileira, vol. 33, no. 1-2, pp. 38-9, 1987.
L. E. Bermudez, Goodman, J., and Petrofsky, M.,
“Role of complement receptors in uptake of Mycobacterium avium by macrophages in vivo: evidence from studies using CD18-deficient mice.”,
Infection and immunity, vol. 67, no. 9, pp. 4912-6, 1999.
L. E. Bermudez, Inderlied, C. B., and Young, L. S.,
“Mycobacterium avium complex in AIDS.”,
Current clinical topics in infectious diseases, vol. 12, pp. 257-81, 1992.
L. E. Bermudez, Parker, A., and Goodman, J. R.,
“Growth within macrophages increases the efficiency of Mycobacterium avium in invading other macrophages by a complement receptor-independent pathway.”,
Infection and immunity, vol. 65, no. 5, pp. 1916-25, 1997.
L. E. Bermudez, Wu, M., and Young, L. S.,
“Interleukin-12-stimulated natural killer cells can activate human macrophages to inhibit growth of Mycobacterium avium.”,
Infection and immunity, vol. 63, no. 10, pp. 4099-104, 1995.
L. E. Bermudez, Young, L. S., and Gupta, S.,
“1,25 Dihydroxyvitamin D3-dependent inhibition of growth or killing of Mycobacterium avium complex in human macrophages is mediated by TNF and GM-CSF.”,
Cellular immunology, vol. 127, no. 2, pp. 432-41, 1990.